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Anemia: Deal breaker to managing Hashimoto’s hypothyroidism

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If you have iron-deficiency anemia, it will be difficult to manage your Hashimoto’s hypothyroidism. Because it robs the cells of oxygen necessary for basic functions, anemia is a deal breaker when it comes to improving your health. Knowing how to identify and address anemia are crucial first steps to managing Hashimoto’s hypothyroidism.

What is iron-deficiency anemia

Although there are many forms of anemia, iron-deficiency is the leading cause of anemia in the United States and the most common nutritional deficiency. The body uses iron to make hemoglobin, a part of blood cells that carries oxygen. When iron is low, the body makes smaller red blood cells and fewer of them. As a result, the body does not get enough oxygen.

Lack of oxygen can increase inflammation and make it more difficult to manage autoimmune conditions such as Hashimoto’s hypothyroidism.

Why oxygen is important when you have Hashimoto’s hypothyroidism

This is a problem because all the body's cells need a constant supply of oxygen to function. All cells have mitochondria, which are like little power plants. The mitochondria produce adenosine triphosphate (ATP), molecules that store and release energy, functioning like rechargeable batteries. This process is necessary to create new tissue, eliminate old tissue, convert food to energy, dispose of waste materials and toxins, and communicate with other cells. Healthy mitochondrial function and ATP production are vital to managing Hashimoto’s hypothyroidism, and they require oxygen to work.

What causes iron-deficiency anemia?

A variety of factors can cause iron-deficiency. They include:

  • Not enough iron in the diet. Iron-rich foods include meat, eggs, and leafy green vegetables.
  • Chronic blood loss in the body from ulcers, heavy bleeding during menstruation, uterine fibroids, hemorrhoids, cancer, or regular aspirin use.
  • Pregnancy. The need for iron grows as the pregnant mother must supply iron for both herself and the growing fetus.
  • Inability to absorb iron. Iron-deficiency is common in undiagnosed celiac disease, which damages and inflames the small intestine so that it cannot absorb nutrients. Other food intolerances or poor digestive function can also result in poor absorption of iron. Studies have shown a strong link between Hashimoto’s hypothyroidism and gluten intolerance.

Symptoms of iron-deficiency anemia

The easiest way to determine whether you have iron-deficiency anemia is through a functional blood chemistry panel, which looks at a complete blood count and iron markers.

You can also evaluate your symptoms to determine whether you may be at risk for iron-deficiency anemia. Symptoms include:

  • Feeling weak and tiring easily
  • Dizziness
  • Feeling grumpy or cranky
  • Headaches
  • Pale skin, nail beds, and gums
  • Short of breath
  • Trouble concentrating

Addressing iron-deficiency anemia for Hashimoto’s hypothyroidism

One should address the root cause of iron-deficiency anemia to better manage Hashimoto’s hypothyroidism. For instance, a gluten-free diet and repairing gut damage resolves anemia in many people. If you need an iron supplement, it's important to choose one that is well absorbed by the body and will not cause an upset stomach or constipation. Ask my office for advice.

Avoid iron toxicity

There are many different forms of anemia besides iron-deficiency anemia, such as anemia caused by a B12 deficiency, inflammation, or an autoimmune disease (pernicious anemia). Supplementing with iron when you don't need it may increase the risk of excess levels of iron in your body. Although the body needs iron to function, in excess it is toxic.

Ask my office for advice on anemia.

Meet the Author

Dr. Matz DC

Dr. Boyle D.A.C.M., LAc., DiplOM. is the founder of the Holistic Wellness Center of the Carolinas where he is the Director of Functional Medicine. He holds a Diplomate in Oriental Medicine and is acupuncture physician and primary care physician in the state of Florida. His post-graduate focus has been in the fields of functional neurology, functional immunology, and functional endocrinology.

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